Food for LIFE! FREE Lunch & Learn

Struggling with Diabetes or other chronic health issue? Looking for natural ways to restore health and vitality? Join me at Magnolia Court where Lucy Richardson, RN and I will share simple ways to improve health through not just food but also by supporting other areas of our life.super mom - super child

This is a FREE event but you MUST RSVP as there will be food and we need to know how many to prepare for. (Be sure to share this with friends and family!)

 

 

You may call or text either: 936-463-0272 or 936-371-9428 to RSVP

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Got Cravings? What to eat instead…

Anyone who has made changes to their diet has probably experienced cravings! I came across a similar chart a few years ago and it has been a huge help when I go through times of craving foods I know are not a healthy choice!!!  Here is my rendition and hope it is beneficial to you in your health journey 🙂

cravings-what-to-eat-instead

Expert Talk: David Perlmutter, MD Brain Inflammation, Autoimmune Disease and Nutrition

I just listened to the first ‘bonus’ video from the Autoimmune Revolution and I was STUNNED! ABSOLUTELY FABULOUS!!!  I am looking forward to the rest of the videos – I have a feeling it is going to end up being a great resource for me on this journey…  If you missed my previous post here is another link below to sign up for the free content!

After registering you will get an email confirmation with a link to the bonus content – The video I just watched was:

Expert Talk: David Perlmutter, MD Brain Inflammation, Autoimmune Disease and Nutrition

(Here is Dr. Perlmutters website http://www.drperlmutter.com/

https://www.thunderclap.it/projects/50763-the-autoimmune-revolution/embed

Betrayal: Autoimmune Disease, what they aren’t telling you…

This is a great series that delves into looking at health conditions through a functional lens, looking at it from a functional perspective 😉

This is an 7 part series which you can sign up here to get the entire series:

https://betrayalseries.com/episode-1-1nyawjkj2?inf_contact_key=7d0cbc282bdddf41ebe8765a8e30e2913dfccc53967e4ef6635564f98c6380b9

Common sense

I recently updated the “nuts & bolts” page where I touch on how our bodies provide ‘medicine’ to heal.  There is a LOT more to put on that page but I wanted to start sharing since there is a wealth of information that I hope to put there…

Anyway, I was reviewing a webinar regarding the cardiovascular system and one more of those ‘stop in my track’ moments occurred.  Most of us have probably heard about the “bad cholesterol” and “plaque” build up.  For those who haven’t – LDL cholesterol is termed the bad guy. Too much of it and it will start sticking to the lining of your blood vessels causing plaque.  High blood sugar and high amount of abdominal fat stimulate the body to release plasminogen activator inhibitor 1 (PAI-1) which promotes blood clotting and build up of plaque.

The “ah ha” moment was because I understand the wonderful function of LDL in our bodies – it is necessary for repair and damage.  For example, do you know what high blood sugar does to the body?  It is like glass or sand which makes abrasions along the cell wall. So when we see high LDL it is a sign that something is in need of repair.  In this example of high blood sugar, in essence our amazing body is protecting itself from ourselves!  The body gets a signal to go repair and protect the wall by forming the plaque.  Of course in the long run this is detrimental, but at least it provides us with the opportunity to make changes. ***

So often the focus is on lowering that LDL number rather than looking as to WHY is that number elevated?   Our bodies make most of the LDL that is measured in blood tests, it is making it for a reason! What is the reason it is making it??? Lowering the LDL doesn’t address the underlying malfunction in our body. Such as maybe its high blood sugar causing damage… Do you know how many diabetics are on statin drugs?  It’s alarming!

Do you now what statin drugs do?  Statins are prescription pills which are very effective at lowering levels of LDL.  Guess what the most common side effect is?  Muscle pain.   Is this surprising??  It shouldn’t be, after all when we take away a necessary repair mechanism from our body what is going to happen? Stuff is gonna start showing the wear and tear.  Not just in our blood vessels, but our entire body!  It is no wonder there is an increase in heart attacks despite the effectiveness of statins lowering LDL.

Common sense tells us things could go very, very wrong!  Fortunately, there are more and more who have decided to take a look at health from a different perspective, one that looks at how the body is supposed to function.  Getting clues from how the body reacts to an adverse environment, letting our body be the guide. Pinpointing the reason behind why the body took evasive action. This kind of digging gets me super excited about health!  Totally lights a fire for me to pursue healthy choices and be in harmony with what my body is telling me!  Am I the only one who feels this way?

Well, gonna get back to the webinar, maybe I’ll get me some veggies to snack on 🙂

{***I added some clarification to my original post as it may not have been clear I was referring to high blood sugar as just an example of damage to our bodies}

Neogenesis… An interesting find for diabetics!

For any type 1 diabetics out there, or anyone interested in some really neat information! I came across this very interesting study that is sure prompting me to have second thoughts!  It addresses the question of whether or not beta cells can regenerate after neonatal time of life… I have only read through this once, but there seems to be some clues that will help put pieces together… Going to go ahead and share this now since it was quite exciting to see that YES the cells CAN regenerate!  Will share more once I dig deeper, in the meantime hope this is helpful 🙂

β-Cell Growth and Regeneration: Replication Is Only Part of the Story
Susan Bonner-Weir, Wan-Chun Li, Limor Ouziel-Yahalom, Lili Guo, Gordon C. Weir, Arun Sharma
Diabetes Oct 2010, 59 (10) 2340-2348; DOI: 10.2337/db10-0084

One point which is very interesting… is the following comment regarding those with gastric bypass surgery… sure makes one consider the role our gut plays in health… And in this particular situation how bypassing impacts blood sugar regulation… What are your thoughts?

“Still more evidence suggesting neogenesis in humans comes from a small group patients who develop hypoglycemia after gastric bypass surgery (79). They have been found to have increased β-cell mass and impressive numbers of islet cells within the ducts (Fig. 2A), accompanied by high circulating levels GLP-1, which has been shown to stimulate neogenesis in rodents (8). From these accumulating circumstantial data, one can be more confident that neogenesis is an important process in humans”.

Are you thinking what I am thinking?  There must be something in the area which was by-passed that is having an adverse effect on insulin production and the islet cells…. So what roadblock did that bypass remove???? Hmmm… is this another connection to my post “Microbiome“?

Studies providing evidence of in vivo neogenesis

Species Observation References
Rat
    Neonates Islet mass growth from cytokeratin+ cells at periphery of islets; β-cell mass determinants measured, and mathematical modeling Bouwens et al. (21); Wang et al. (89); Finegood et al. (5); Scaglia et al. (6); Bonner-Weir et al. (22)
    Neonatal STZ Appearance of small islet clusters associated with ducts Portha and colleagues (90)
    Duct ligation Appearance of small islet clusters associated with ducts Edstrom (50)
Wang et al. (32)
    Duct ligation + gastrin Appearance of small islet clusters associated with ducts Rooman et al. (52)
    Pancreatectomy (90%) New lobe formation with new islets, enhanced replication too Bonner-Weir and colleagues (31, 38)
    Zucker fatty and Zucker fatty diabetic Increased small islet clusters associated with ducts Pick et al. (91)
    Zucker fatty rats Increased small islets associated with ducts, increased number islets Jetton et al. (44)
    20% glucose infusion (48 h) Increased small islet clusters associated with ducts Jetton et al. (92)
    Exendin 4 Increased hormone+ cells in ducts Xu et al. (8)
  Soybean trypsin inhibitor Increased volume of insulin+ cells in ducts Weaver et al. (33)
Mouse
    Early postnatal Increased number of islets from 1 week to 2 months; lineage tracing of duct-specific promoter birth to 4 weeks Inada et al. (20); Peng et al. (26)
    Ductal ligation Ngn3+ cells in and adjacent to ducts Xu et al. (19)
CAIICreERT lineage tracing Inada et al. (20)
    Alloxan perfusion to part of pancreas Replication in nonperfused part and neogenesis in perfused Waguri et al. (34)
    Alloxan + betacellulin Increased ICC/mm2 pancreas Yamamoto et al. (93)
    Alloxan + adv-betacellulin Significantly increased insulin+ duct cells Tokui et al. (94)
    Alloxan + EGF and gastrin Significantly increased insulin+ duct cells Rooman and Bouwens (95)
    Pancreatectomy (60%) Increased small clusters before β-cell proliferation; FOXM1 necessary for proliferation but for not neogenesis Peshavaria et al. (46); Ackermann Misfeldt et al. (47)
    Retroductal adv-GFP (neonatal) GFP in islets over first 2 weeks Peng et al. (26)
    Metallothionen:TGF-α × Ins:gastrin Insulin+ cells in metaplastic ducts, increased islets Wang et al. (37)
    RIP:interferon-ψ Increased insulin+ clusters in ducts Gu and Sarvetnick (36)
    NeuroD-null mice 10% duct in null mice had “budding” insulin+ cells Huang et al. (96)
    Glucagon:Pax4 Reprogramming of α- to β-cells and replenishment of α from ducts Collombat et al. (30)
Pig
    Obese minipig, after 1 of year age Increased islet numbers but same mean volume of islets Larsen et al. (14)
Human
    Autopsied pancreas, birth to age 20 years After 12 years, most 0.5–1.2% insulin+ duct cells, some none Meier et al. (62)
    Donor pancreas, aged 7–70 years Unchanged low level of neogenesis from 7 to 70 years Reers et al. (65)
    Autopsied, control subjects Obese 1.2% insulin+ duct cells but lean 0.6 ± 0.2% insulin+ duct cells Meier and colleagues (66, 74)
    Autopsied, chronic pancreatitis Significantly increased glucagon+ or insulin+ duct cells Phillips et al. (76)
    Biopsied failed pancreatic transplant Increased insulin+ ducts in transplants with recurrent autoimmunity Martin-Pagola et al. (78)
    Pregnancy Increased β-cells with no change in replication, cell size, or apoptosis frequency; increased insulin+ duct cells and very small islets Butler et al. (15)

Chart is from the above study and can be found at this link.

Would love to hear your thoughts!